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Strikingly, the absence of Gclc in FoB resulted in secretory properties that recapitulated those of Gclc -sufficient MZB at both the transcriptional and protein levels (Supplementary Fig

Moreover, as a key regulator of ferroptosis, GPX4 and its role in OA have been demonstrated in three recently published studies [22, 23, 111]

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Of note, transcription of cyclin-dependent kinase inhibitor, p21 ( CDNK1A ) was significantly upregulated in response to GSTO1 knockdown while that of the cell cycle regulatory proteins cyclin D1 ( CCND1 ), cyclin E1 ( CCNE1 ), as well as Myc were found downregulated (GEO accession No