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Effect of thioltransferase (glutaredoxin) deletion on cellular sensitivity to oxidative stress and cell proliferation in lens epithelial cells of thioltransferase knockout mouse

The combined effects of increased cytoplasmic monoamine availability and transporter phosphorylation cause reversal of transporter flux, resulting in non-exocytotic neurotransmitter efflux into the synaptic cleftthe characteristic one-two punch mechanism underlying amphetamine-induced monoamine release

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