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Mitochondrial GR is essential for: Protecting the electron transport chain components from oxidative damage Maintaining mitochondrial DNA integrity against oxidative lesions Supporting ATP production by preserving mitochondrial membrane potential Preventing mitochondrial-initiated apoptosis triggered by excessive oxidative stress Facilitating mitochondrial glutathione transport and utilization The mitochondrial glutathione system is particularly critical in neurons and other cells with high energy demands, where mitochondrial dysfunction can have catastrophic consequences

The field of metal-based composites research took off in the 1970s when scientists tried combining metals and alloys with ceramic reinforcements through powder metallurgy, rheocasting, and stir casting, all of which produced intriguing outcomes [45, 46]

Subsequent studies also confirmed that DMB can improve certain diseases by reducing TMAO concentrations, such as allogenic gain-versus-host disease (Wu et al., 2020) and endothelial dysfunction (Brunt et al., 2020)

These findings provide strong rationale for the ongoing Phase IIb trials investigating simplified weekly and monthly dosing regimens