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Rather than indiscriminately blocking macrophage activity (which would impair the early inflammatory debridement phase of wound healing), GHK-Cu research documents a shift in macrophage phenotype from pro-inflammatory M1 toward the anti-inflammatory, pro-healing M2 profile at later stages of the inflammatory response

Antioxid Redox Signal (2006) 8(910):186579.10.1089/ars.2006.8.1865 7 PaulsenCECarrollKS

Therefore, these mice can be used to study the impacts of disorders in this system

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